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ISSN: 1920-3799

The Orgonomic Theory of Cancer

by Correa, Paulo N. & Correa, Alexandra N.
Aurora Biophysics Research Institute

J Biophys Hematol Oncol, Volume 1, Issue 3 (March 2010),  pp. 1-41

Article ID:   JBHO01-03-01

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The Journal of Biophysics, Hematology and Oncology is an open-review journal. If you wish to contribute a review of this article, please send your review to   editor.JBHO@aetherometry.com .


We examine in detail Wilhelm Reich's orgonomic theory of the etiology of cancer and his model of the cytological stages of cancer. The orgonomic model of cancer stands alone in linking the persistence and intensification of social and psychosomatic factors that negatively affect emotional expression with the onset of hypoxic conditions, local and systemic, that select for malignant phenotypes. The rarely broached main novelty of the model suggests that every manifestation of somatic cancer is preceded by increasing RBC dysfunctionality, and that the leukocytosis characteristic of most chronic leukemias is in fact an auto-immune reaction geared against RBC fragments and their (self-)antigens. Even though unproven, these are provocative suggestions that could turn out to be substantially correct. The same cannot be said for the orgonomic model's contention that the real cancer cells are amoeboid cells that arose de novo (heterogenically) from the vesicular byproducts of one or more dying or decaying tissue cells - or for Reich's proposed staging of cancer. In this context, the vesicle-based concept of a PA bion is found to be wanting and unable to acquire a functional sense - as it is employed to designate such widely different biological phenomena as to become merely a catch-all. In contrast, Reich's identification and isolation of his 'T-bacilli', despite some discrepancies, seems to largely coincide with the identification of mycoplasma. His proposed role for these T-bacilli in chronic inflammation and his experimental induction of leukemoid disease in mice injected with T-bacilli isolates are coadunate with current views of the possible role of mycoplasma in the co-induction of leukemia or leukemoid states.